Clean Diesel Vehicle Sales up 27.5 Percent

According to information compiled by consultancy firm Baum & Associates, sales of clean diesel vehicles in the U.S. jumped by 27.5 percent during the first half of 2012.

Diesel sales have seen incremental increases in the last two years, witnessing double-digit gains in 20 out of the last 23 months. Although diesel volume in the U.S. still lags far behind that of Europe, where oil burning cars account for more than 50 percent of the market, the gains represent very significant ones, especially for a nation where, less than a decade ago, diesel was still considered a dirty word by many motorists.

Diesel volume is expected to increase significantly in the U.S., with new models introduced as automakers strive to meeting tightening Corporate Average Fuel Economy standards, which given current rules, will require a fleet-wide wide “54.5” miles per gallon by 2020. In fact, data from Pike Research indicates that diesels will make up 12.4 percent of the market by 2018.

Among the oil burning vehicles expected within the next 18-24 months included a Cadillac ATS diesel, Chevy Cruze diesel, Ford Transit Connect diesel, Jeep Grand Cherokee and Mazda SkyActiv-D.

Updated versions of diesel cars previously sold here are also on the way, including a new generation VW Beetle TDi and Mercedes S-Class BlueTec.

Autoguide


  • Anonymous

    As I said before, there are no “clean diesels”. On the contrary diesels are carcinogenic and cause several type of cancers.

  • ACAgal

    Anonymous, you are 100% correct!

  • KMCoates

    Anonymous & ACAgal,
    Get a grip. Unless you’re walking, anything you drive or use for transport is creating some emissions either from the tailpipe or upstream or in its manufacturer. Clean diesels are comparable to gas or alternative fuel-powered vehicles. They deliver excellent fuel economy and low CO2 emissions, which is part of why their popularity is growing.

  • ACAgal

    If you aren’t used to med terms: lipid translates to fat in ordinary English
    The following study means diesel really presents problems, to other organs in addition to the lungs as referred to in ultra fine pollution, and is only one of many that implicate diesel, as a factor, in more than pulmonary and heart health issues

    Abstract
    1-Nitropyrene (1-NP) is a nitro-polycyclic aromatic hydrocarbon (nitro-PAH) present in diesel exhaust and bound to particular matter in urban air. We show that 1-NP and the referent PAH benzo(a)pyrene (BP) induce apoptosis and a lipid accumulation dependent on cytochrome P450 1A1-metabolites in mouse hepatoma cells, whereas 1-amino-pyrene had no effect. The caspase inhibitor, N-benzyloxycarbonyl-Val-Ala-Asp(O-Me) fluoromethyl ketone (Z-VAD-fmk), inhibits 1-NP-induced apoptosis, but failed to alter 1-NP-triggered lipid accumulation determined by Nile red staining. We further show that cholesterol and fatty acid contents are modified after nitro-PAH exposure and that 1-NP-induced cholesterol level is partially involved in related apoptosis. In parallel, the activity of the stearoyl-CoA desaturase 1 (SCD1), determined by fatty acid analysis, and its expression are reduced by 1-NP. The role of SCD1 in 1-NP-induced apoptosis is demonstrated in cells down-expressing SCD1, in which an increased apoptosis is observed, whereas the SCD1 overexpression elicits the opposite effects. In contrast, changes in SCD1 gene expression have no effect on the induced lipid accumulation. Moreover, 1-NP increases the activity of the AMP-dependent protein kinase (AMPK) leading to a caspase-independent apoptosis. Overall, our study demonstrates that the 1-NP-induced apoptosis is caspase- and AMPK-dependent, and is associated to a decrease of SCD1 expression which results in an alteration of lipid homeostasis.
    Copyright © 2011 Elsevier Ireland Ltd. All rights reserved.
    PMID: 21872649 [PubMed - indexed for MEDLINE]
    Publication Types, MeSH Terms, Substances

    Source info:
    Toxicol Lett. 2011 Oct 30;206(3):289-99. Epub 2011 Aug 18.
    Mechanisms involved in lipid accumulation and apoptosis induced by 1-nitropyrene in Hepa1c1c7 cells.
    Podechard N, Tekpli X, Catheline D, Holme JA, Rioux V, Legrand P, Rialland M, Fardel O, Lagadic-Gossmann D, Lecureur V.
    Source
    EA4427-SeRAIC, Team Toxicity of Polycyclic Aromatic Hydrocarbons, IFR140, Université de Rennes 1, Faculté des Sciences Pharmaceutiques et Biologiques, IRSET, France.

  • wxman

    ACES Phase 1 study reported that exhaust levels of nitroPAH from 2007-compliant truck diesel engines were barely detectable and only slightly above that of filtered dilution tunnel air (Phase 1 of the Advanced Collaborative Emissions Study, June 2009, Coordinating Research Council, http://www.crcao.org/reports/recentstudies2009/ACES%20Phase%201/ACES%20Phase1%20Final%20Report%2015JUN2009.pdf).

    ACES Phase 3 study reported that animals exposed to concentrated exhaust from a 2007-complaint diesel engine for 16 hours per day, 5 days per week, for up to 12 months have experienced virtually no health effects (http://pubs.healtheffects.org/getfile.php?u=708). The full report also shows that particle emissions were below detection limits except during DPF regeneration (http://pubs.healtheffects.org/getfile.php?u=709, Figure 2, page 19)

    Many studies, including one by CARB (NOT a diesel proponent) have shown that particulate emissions are indistinguishable from background levels. The particulate emissions are at least 2 orders of magnitude lower than gasoline engines, and gasoline engines have been shown to have particle emissions which EXCEED those of “old tech” diesel engines (i.e., unfiltered) under some common driving conditions (rapid acceleration, high speeds).

    Opposition to diesel technology on health/environmental grounds is no longer legitimate.

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